exam-11-this-exam-has-a-total-of-18-points-each-point-corresponds-to-a-numbered-question

This exam has a total of 18 points. Each point corresponds to a numbered question. The numbered question is to be answered. The correct answer to a numbered question will give you one point. If the answer is partially correct, partial credit will be given as a decimal value. If the answer is incorrect, no credit will be given. If the question is unanswered, no credit will be given so it is better to guess than to leave a question blank. The number of points earned will be divided by the total number of points on the exam and multiplied by 100 to get your grade. Good luck!

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  1. In the background section the authors wrote “It was hypothesized that the solitary presence, and not the amount of blood, could be responsible for the severe expansion of the secondary damage.” In terms of the solitary presence, is this a null hypothesis or an alternate/alternative hypothesis? (1 point)

  1. How do you know that your answer to question 1 is correct? (1 point0

  1. In the background section the authors wrote “It was hypothesized that the solitary presence, and not the amount of blood, could be responsible for the severe expansion of the secondary damage.” In terms of the amount of blood, is this a null hypothesis or an alternate/alternative hypothesis? (1 point)

  1. How do you know that your answer to question 3 is correct? (1 point)

  1. In the background section the authors wrote “Argatroban derives from the amino acid arginine and binds to the active catalytic binding site of thrombin.” Based on this quote, what is argatroban? (1 point)

  1. How do you know that your answer to question 5 is correct? (1 point)

  1. In the background section the authors wrote “Argatroban derives from the amino acid arginine and binds to the active catalytic binding site of thrombin.” Based on this quote, what is thrombin? (1 point)

  1. How do you know that your answer to question 7 is correct? (1 point)

  1. In the background section the authors wrote “Direct thrombin blocking is also possible with Bivalirudin …” Based on this quote what is Bivalirudin? (1 point)

  1. How do you know that your answer to question 9 is correct? (1 point)

  1. In the background section the authors wrote “A small portion of thrombin and prothrombin is expressed in neural cells [11], and this is associated with chronic neurodegenerative diseases like Alzheimer [12], Parkinson [13] and Multiple Sclerosis [14].” Why is thrombin associated with Multiple Sclerosis? (1 point)

  1. Quote something from the article as evidence that your answer to question 11 is correct. (1 point)

  1. In the background section the authors wrote “We hypothesize that the acute release of thrombin from the subdural hematoma could be one of the harmful blood constituents, which trigger long-term lesion enlargement through early activation of PARI.” Is this hypothesis a null hypothesis or an alternate/alternative hypothesis? (1 point)

  1. How do you know that your answer to question 13 is correct? (1 point)

  1. In the lesion volume 14 days after ASDH section of the results section the authors wrote “PARI blockage resulted in the following lesion volumes: Animals treated with vehicle had an average tissue loss of 6.5 ±2.0 mm3 with one µg SCH-79797 lesion volume was 4.3 ±0.7 mm3and with five µg SCH-7979 the average damage was 3.8 ±1.1 mm3 (all p: n.s., Fig. 2b).” Based on this quote, why was there tissue loss? (1 point)

  1. Did this study prove that thrombin is one of the key players in modulating secondary damage after ASDH? (1 point)

  1. How do you know that your answer to question 16 is correct? (1 point)

  1. In the discussion section the authors wrote “Thrombin influences signal transduction in the brain predominantly via PARI [48, 49].” What is the role of PARI in signal transduction? (1 point)

 
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